Prevalence of Helicobacter pylori vacA, cagA and iceA genotypes and correlation with clinical outcome
Toxins | Free Full-Text | Helicobacter pylori BabA–SabA Key Roles in the Adherence Phase: The Synergic Mechanism for Successful Colonization and Disease Development
Helicobacter pylori Infection and Gastric Cancer — Is Eradication Enough to Prevent Gastric Cance | IntechOpen
What exists beyond cagA and vacA? Helicobacter pylori genes in gastric diseases
Schematic hypothesis presenting model of Hp positive cagA and vacA... | Download Scientific Diagram
Synergistic and antagonistic interactions between VacA and CagA.(A)... | Download Scientific Diagram
Occurrence of cagA+ vacA s1a m1 i1 Helicobacter pylori in farm animals in Egypt and ability to survive in experimentally contaminated UHT milk | Scientific Reports
Helicobacter pylori Infection Modulates Host Cell Metabolism through VacA-Dependent Inhibition of mTORC1 - ScienceDirect
PDF) Helicobacter pylori cagA and vacA genes in dyspeptic Ghanaian patients | Edwin Wiredu - Academia.edu
Helicobacter pylori virulence factor CagA promotes tumorigenesis of gastric cancer via multiple signaling pathways | Cell Communication and Signaling | Full Text
Helicobacter pylori VacA, a paradigm for toxin multifunctionality | Nature Reviews Microbiology
Helicobacter pylori Employs a Unique Basolateral Type IV Secretion Mechanism for CagA Delivery - ScienceDirect
The VacA and The CagA of Helicobacter Pylori: Two Multitasking Proteins of a Multitasking Bacterium
Frontiers | Alterations in Helicobacter pylori Triggered by Contact with Gastric Epithelial Cells
Helicobacter pylori CagA: From Pathogenic Mechanisms to Its Use as an Anti-Cancer Vaccine | Semantic Scholar
Frontiers | A Tale of Two Toxins: Helicobacter Pylori CagA and VacA Modulate Host Pathways that Impact Disease
Helicobacter pylori virulence factor CagA promotes tumorigenesis of gastric cancer via multiple signaling pathways | Cell Communication and Signaling | Full Text
Helicobacter pylori promote inflammation and host defense through the cagA‐dependent activation of mTORC1 - Feng - 2020 - Journal of Cellular Physiology - Wiley Online Library